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Aug 23, 2020 
 Asprise OCR v4.0.3.0 Cracked.rar
Asprise OCR v4.0.3.0 Cracked asprise ocr v4.0.3.0 With Crack. 1 Crack is working. rar
Asprise OCR v4.0.3.0 Crack asprise ocr v4.0.3.0 With Crack. 1 Crack is working. rar
Asprise OCR v4.0.3.0 Crack asprise ocr v4.0.3.0 With Crack. 1 Crack is working. rar
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Overview
Trauma patients with acute pain (for example, following surgery) may develop hyperalgesia (an increased response to painful stimuli). This pain is thought to result from the excessive release of neuro-chemicals in the pain pathway. Hyperalgesia may be acute or chronic. Hyperalgesia following injury to the brain is called central sensitization, and is thought to underlie chronic pain. Research is showing that chronic hyperalgesia is a contributing factor in pain associated with fibromyalgia, irritable bowel syndrome, migraine and depression.
Disclaimer:
These summary points have been derived from a variety of sources, and are not necessarily drawn from the most current of scientific knowledge.
The brain itself is not pain-free; it has its own pain pathways.
Pain is processed in the thalamus and sensory cortex and is mediated by various neurotransmitters, such as glutamate, and neuropeptides, such as substance P, CGRP, encephalin, dynorphin, and calcitonin gene-related peptide (CGRP).
Pain is a reflex action caused by inflammation and injury. Pain is processed in the brain and then sent to the spinal cord.
The brain has special cells that respond to pain. These are called "pain neurons," and are activated when a painful stimulus is perceived.
Pain can be perceived through touch. This is called tactile stimulation. Pain can also be perceived through other modalities. For example, stimulation of the eyes or throat can activate the pain pathways.
Cancer patients often have chronic hyperalgesia.
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