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Hyperthyroidism is a potentially life-threatening disorder in which hyperthyroidism results from the overproduction of thyroid hormone by the thyroid gland. Radioiodine (131I) therapy, especially when given as a single dose, remains the most effective form of therapy for the majority of patients with hyperthyroidism. However, it is associated with many risks, including the induction of thyroid cancer. Subsequently, several less-intrusive approaches have been adopted. These include the use of antithyroid drugs, particularly propylthiouracil (PTU) and methimazole (MMI), which are more convenient and less toxic than 131I therapy. Second-generation thionamides (methimazole and carbimazole), are generally recommended as first-line therapy in the United States and Europe, but are contraindicated in areas with endemic iodine deficiency. Carbimazole should be reserved for patients unable to take PTU or MMI, patients with significant cardiovascular disease or known allergy to propylthiouracil, and those who are prone to malignancy or who have thyroid cancer. Third-generation thionamides (phenylbutylnitrate, glypressin, or tiamenidine) have the advantage of not being associated with transient antithyroid effects and should be considered in patients who are currently treated 3125841983
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